Mechanism of Action of the Presynaptic Neurotoxin, Tetanus Toxin
Abstract
During the early phase of this project we were successful in establishing a cultured cell model system, the PC12 pheochromocytoma cell line, to study the mechanism of action of tetanus toxin. Further we have studied the characteristics of the intoxication pathway (Sandberg et al. 1989) and have found that it is analogous to that which has been characterized, to some extent, in vivo (Simpson, 1986; Habermann and Dreyer, 1986). The major thrust during the past year was to exploit this well characterized model system to gain insight into the molecular mechanism of action of tetanus toxin. In the present study, we have utilized a preparation of permeabilized, NGF-differentiated, PC12 cells to examine the role of cGMP in neurotransmitter release. An important finding is that cGMP can stimulate neurotransmitter release from such cells in a Ca+2- independent manner. Further, NGF-differentiated PC12 cells show two phases of vesicular neurotransmitter release that can be distinguished not only by their differential sensitivity to Ca2+, but also in their sensitivity to cGMP.
Document Details
- Document Type
- Technical Report
- Publication Date
- Apr 30, 1990
- Accession Number
- ADA243277
Entities
People
- Terry B. Rogers
Organizations
- University of Maryland School of Medicine