The Fibroblast Response to Anoxia: A Potential Direct Role in Wound Healing.

Abstract

Cellular anoxia constitutes an environmental stimulus experienced within early stage healing wounds. Anoxia has effects on fibroblasts which make important contributions to regulating the progressive events of wound healing. These effects are mediated through specific gene induction, resulting in activation of a cellular program including enhanced glycolysis, protease secretion, endonuclease production, and expression of an angiogenesis factor. In a delayed response, fibroblast contractile processes activate at a time appropriate for wound contraction. The anoxic fibroblast response is constitutively activated in a large fraction of malignancies, where it contributes to invasiveness and genomic instability.

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Document Details

Document Type
Technical Report
Publication Date
Dec 01, 1994
Accession Number
ADA291344

Entities

People

  • Garth R. Anderson

Organizations

  • Roswell Park Comprehensive Cancer Center

Tags

DTIC Thesaurus Topics

  • Alcohols
  • Biodegradable Plastics
  • Biological Factors
  • Biomedical And Dental Materials
  • Cancer
  • Cells
  • Chemistry
  • Debridement
  • Fibroblasts
  • Genomic Instability
  • Macrophages
  • Neoplasms
  • Polymeric Films
  • Polyvinyl Alcohol
  • Proteins
  • Tissues
  • Wound Healing

Readers

  • Cardiovascular Physiology
  • Molecular Biology and Genetics
  • Trauma Surgery or Emergency Medicine.