Mechanisms Associated with the Toxicity of Polyhalogenated Cyclic Hydrocarbons and Heavy Metals.

Abstract

We have hypothesize that the toxic manifestations of polyhalogenated cyclic hydrocarbons and heavy metals are at least in part induced through a series of events involving the production of reactive oxygen species, activation of the protein kinase C system and release of tumor necrosis factor-a (TNFa). The sequence of these events is not known, and the relationship between these events and the induction of stress protein has not been studied. Both in vivo and in vitro effects on these parameters as well as acute and chronic studies will be determined. The above hypothesis will be tested by completing the following specific aims. Excellent progress has been made relative to the specific objectives of this research project. Based on the results which have been completed during the past three years, 21 manuscripts were published or are in press in refereed journals, and 14 presentations have been made at national and international scholarly meetings.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 1996
Accession Number
ADA325697

Entities

People

  • Sidney J. Stobs

Organizations

  • Creighton University

Tags

DTIC Thesaurus Topics

  • Acetaldehyde
  • Air Force
  • Aldehydes
  • Alkenes
  • Apoptosis
  • Biological Factors
  • Biological Products
  • Cell Physiological Processes
  • Cyclic Hydrocarbons
  • Formaldehyde
  • Free Radicals
  • Heavy Metals
  • Hydrocarbons
  • Liquid Chromatography
  • Oxidative Stress
  • Programmed Cell Death
  • Toxicology

Readers

  • Immunology and Pathology
  • Organic Chemistry
  • Technical Research and Report Writing.