Chimeric Enzyme/Prodrug Therapy as Novel Gene Therapy for Breast Cancer

Abstract

We have constructed a series of artificial chimeric genes each composed of a tumor-associated CD44 alternative splicing unit (ASU) which includes alternatively spliced exons and flanking introns and exons and the cytosine deaminase (CD) gene. The selective expression of active CD44/CD chimeric protein potentially enables tumor-specific killing via the RNA metabolism of alternative splicing following administration of the prodrug 5-FC. Insertion of the CD44 transmembrane region between 5'CD44 encoding exons and 3'-CD cDNA anchors the chimeric protein to the cell membrane while partitioning CD44 to the extracellular and CD to the intracellular compartments, respectively and results in no significant loss of CD enzymatic function. This modification makes it possible to introduce specific therapeutic receptor or antigen sites located outside the membrane allowing for recognition of cancer cells surviving the toxicity of the 5-FU anabolite.

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Document Details

Document Type
Technical Report
Publication Date
Aug 01, 1997
Accession Number
ADA338689

Entities

People

  • David L. Cooper

Organizations

  • University of Pittsburgh

Tags

DTIC Thesaurus Topics

  • Breast Cancer
  • Cell Membrane
  • Cells
  • Chemistry
  • Cytosine
  • Efficiency
  • Gene Therapy
  • Genetic Code
  • Laboratory Animals
  • Materials
  • Membranes
  • Metabolism
  • Molecules
  • Mrna
  • Neoplasms
  • Proteins
  • Therapy

Fields of Study

  • Biology

Readers

  • Forest Ecology
  • Molecular Genetics
  • Oncology (Cancer Research).

Technology Areas

  • Biotechnology
  • Biotechnology - Cancer Biotech