Estrogen Receptor-Mediated Transcription In Vitro

Abstract

Estrogen receptor (ER) is a ligand activated transcription activator. The detailed mechanism of ER mediated transcription is unclear. We demonstrated that ER mediated transcription in cell free transcription system is ligand dependent. Antiestrogens ICI 164,384, ICI 182,780 and 4-hydroxytamoxifen significantly inhibited ER mediated transcription. Estradiol overcame the inhibitory effect of the antiestrogens and induced ER mediated transcription. Under the condition used for the transcription assay, ICI164,384 and ICI182,780 inhibited ER-ERE complex formation which might contribute to the inhibitory effect of these antiestrogens on ER mediated transcription. 4 OH TAM changed the mobility of ER ERE complex which suggests a conformational change of the complex. Steroid receptor coactivator 1 (SRC- 1) significantly augmented ER mediated transcription in vitro, supporting that SRC-1 is involved in ER signal pathway. ER hormone binding domain (HBD) that binds to ER associated proteins (ERAPs) in a ligand dependent manner in previously studies inhibited ER mediated transcription in vitro. In contrast, truncated ER HED lacking of 535 to 595 amino acids which binds to estradiol but does not associate with ERAPs did not affect ER Mediated effect in this study, suggesting that ERAPs are required for the full transactivation activity of ER.

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Document Details

Document Type
Technical Report
Publication Date
Dec 01, 1997
Accession Number
ADA340593

Entities

People

  • Hong Liu
  • Myles A. Brown

Organizations

  • Dana–Farber Cancer Institute

Tags

DTIC Thesaurus Topics

  • Acids
  • Amino Acids
  • Baculoviridae
  • Biomedical Research
  • Breast Cancer
  • Cell Physiological Processes
  • Cells
  • Chemical Compounds
  • Chemistry
  • Deoxyribonucleic Acids
  • Estrogens
  • Hormones
  • Laboratory Animals
  • Materials
  • Proteins
  • Recombinant Dna
  • United States

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.