Mechanism of Thyroid Hormone-Induced Osteoporosis.

Abstract

The goal of the research is to determine the mechanism by which thyroid hormone (T3) affects bone, and the cause of the osteoporosis that can result when T3 concentrations are excessive. Our previous publications had shown that T3 increases the production of the bone growth factor insulin-like growth factor-I (IGF-I) in osteoblastic cells and promotes the production of the osteoclastogenic cytokine interleukin-6. The experiments carried out during the first grant year evaluated models that could be used to study the roles of these local factors in the anabolic and resorptive effects of T3 on bone and the molecular mechanisms by which T3 stimulated their production. The early findings using antibodies to the IGF-I and IL-6 receptors and antisense oligonucleotides to the IGF-I receptor support a role for these factors in the thyroid hormone effects. Studies at the level of gene expression show T3 augmentation of IL-6 production in human and rodent osteosarcoma cells, a lack of effect of T3 on IL-i receptor expression, and T3 potentiation of parathyroid hormone-induced IL-6 mRNA production.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 1997
Accession Number
ADA340690

Entities

People

  • Paula H. Stern

Organizations

  • Northwestern University

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Antibodies
  • Antisense Elements (Genetics)
  • Bone Diseases
  • Cell Line
  • Cells
  • Chemistry
  • Gene Expression
  • Growth Factors
  • Hormones
  • Materials
  • Osteoblasts
  • Osteoporosis
  • Production
  • Proteins
  • Sarcoma
  • Thyroid Hormones
  • Tissue Extracts

Fields of Study

  • Medicine

Readers

  • Immunology and Pathology
  • Molecular Biology and Genetics
  • Toxicology/Environmental Toxicology