Role of MYC in Anchorage-Dependent Growth

Abstract

When deprived of adhesion to a solid substrate, fibroblasts are unable to proliferate and arrest in the G1 phase of the cell cycle. We have shown that there is a similar adhesion -responsive (G1 restriction point in the immortalized human mammary epithelial cells line, 184A1N4. Unlike normal cells, transformed cells gain the ability to bypass this integrin-mediated control of proliferation and to grow in an anchorage-independent manner. We are examining the effect of c-Myc overexpression, which frequently occurs in breast cancer, in the adhesion-dependent regulation of the Gl-S phase transition.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 1998
Accession Number
ADA364661

Entities

People

  • Christelle M. Benaud

Organizations

  • Georgetown University

Tags

DTIC Thesaurus Topics

  • Adhesion
  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Degradation
  • Epithelial Cells
  • Growth Factors
  • Inhibitors
  • Laboratory Animals
  • Materials
  • Membranes
  • Molecules
  • Neoplasms
  • Proteins
  • Regulations

Fields of Study

  • Biology
  • Medicine

Readers

  • Molecular Biology and Genetics