Characterization of a B-Catenin-Associated Kinase.

Abstract

Several lines of evidence suggest that accumulation of cytoplasmic Beta-catenin transduces an oncogenic signal. We show that Beta-cantenin is ubiquitinated and degraded by the proteosome and that Beta-cantenin stability is regulated by a diacylglycerol-independent protein kinase C-like kinase activity which is required for Beta-catenin ubiquitination. We also define a six amino acid sequence found in both Beta-catenin and the NF-kB regulatory protein IkappaBalpha which, upon phosphorylation, targets both proteins for ubiqultination. Mutation of a single serine within the ubiquitin targeting sequence (UTS) prevents ubiquitination of Beta-catenin. Mutations within the UTS of Beta-catenin may be oncogenic.

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Document Details

Document Type
Technical Report
Publication Date
Aug 01, 1998
Accession Number
ADA375192

Entities

People

  • Stephen W. Byers

Organizations

  • Georgetown University

Tags

DTIC Thesaurus Topics

  • Amino Acids
  • Biomedical And Dental Materials
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemical Reactions
  • Chemical Synthesis
  • Chemistry
  • Colon Cancer
  • Cytoskeleton
  • Enzymes
  • Growth Factors
  • Kinases
  • Materials
  • Mutations
  • Neoplasms
  • Proteins

Fields of Study

  • Biology
  • Computer science

Readers

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