Pyridostigmine-Induced Neurodegeneration: Role of Neuronal Apoptosis.

Abstract

Pyridostigmine causes apoptotic cell destruction in rat brain. The present study reveals mechanisms by which pyridostigmine may cause neuronal death. Pyridostigmine-induced apoptosis is mediated by at least two different receptors in cell cultures from brains of rat pups. Pretreatment of cerebellar granule cells with muscarinic or NMDA receptor blockers attenuates pyridostigmine-induced apoptosis. Both these receptors also mediate increases in reactive oxygen species and calcium in granule cells exposed to pyridostigmine. Elevated intracellular calcium appears to lead to formation of reactive oxygen species since generation of free radicals is inhibited in cells in calcium free media. The reactive oxygen species in turn appear to initiate the apoptotic cell death process, since antioxidants decrease cell destruction by pyridostigmine. These in vitro results suggest that oxidative stress is a factor which causes apoptotic brain cell destruction in certain individuals after pyridostigmine administration.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 1999
Accession Number
ADA377168

Entities

People

  • Gary Isom

Organizations

  • Purdue Research Foundation

Tags

DTIC Thesaurus Topics

  • Acetylcholinesterases
  • Acids
  • Antioxidants
  • Apoptosis
  • Blood
  • Brain
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Culture Techniques
  • Demographic Cohorts
  • Free Radicals
  • Inhibition
  • Microscopes
  • Oxidative Stress
  • Programmed Cell Death
  • Seizures

Fields of Study

  • Biology

Readers

  • Molecular Biology and Genetics
  • Neuroscience
  • Neurotoxicology