Contribution of the Receptor/Ligand Interaction Between CD44 and Osteopontin to Formation of Breast Cancer Metastases
Abstract
Splice variants of the homing receptor CD44 may mediate metastasis formation by various tumors through interaction with the cytokine osteopontin which mediates cell attachment or migration. We have identified the mechanism of cell motility as a two-step process involving both major osteopontin receptors. We have further characterized the important involvement of osteopontin in delayed type immune responses which may have implications for tumor immunology. Future experiments will define CD44-dependent homing and invasion in vivo using our recently generated stably transfected tumor cells of various CD44 splice variants.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 1999
- Accession Number
- ADA384133
Entities
People
- George F. Weber
Organizations
- Dana–Farber Cancer Institute