Targeted Therapy of Human Breast Cancer by 2-5A-Antisense Directed Against Telomerase RNA

Abstract

In most normal cells DNA is lost from the ends of the chromosomes (telomeres) at each cell cycle. In rapidly growing tumor cells this eventually results in chromosome instability and cell death. To overcome this problem most cancer cells re-express the telomerase enzyme which prevents telomere erosion. To establish whether expressing telomerase is essential to maintain the malignant phenotype in these tumor cells, we have used an anti sense oligonucleotide approach targeting the RNA component of the telomerase enzyme. The oligonucleotides used carry a 2-5A moiety which activates RNAseL to selectively destroy the target RNA. Using this system we have shown that targeting telomerase causes rapid cell death in several different breast cancer cell lines in vitro. This cell death is not seen when the cells are treated with control oligos carrying mismatches in the target sequence. Cell death is due to apoptosis.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 1999
Accession Number
ADA384610

Entities

People

  • John K. Cowell

Tags

DTIC Thesaurus Topics

  • Antisense Elements (Genetics)
  • Apoptosis
  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chromosomes
  • Endothelial Cells
  • Epithelial Cells
  • Genetics
  • Laboratory Animals
  • Materials
  • Molecules
  • Neoplasms
  • Programmed Cell Death
  • Targeting
  • Targets

Fields of Study

  • Biology

Readers

  • Molecular Genetics
  • Oncology
  • Oncology and Biomarker-Based Cancer Detection.