HRAD51 Involvement in Genomic Instability and Development of Breast Cancer

Abstract

It becomes increasingly clear that both BRCA1 and BRCA2 tumor suppressor genes are involved in hRAD51-dependent DNA repair by homologous recombination. In this study, we will test whether DNA recombinational repair may be compromised during breast cancer development. We first examined the hRAD51 gene in tumors with 15q14-15 deletions. We did not find any changes compared to normal tissue which supports (among other possibilities, see accompanying reprint) the notion that hRAD51 is an essential gene. We have also examined interactions among members of the hRAD51 protein family and between hRAD51 family members and BRCA1/2. All hRAD51 homologs seem to exist in a multiprotein complex (hRAD51 proteome), and examination of the functional significance of these interactions is currently in progress.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2000
Accession Number
ADA385431

Entities

People

  • Richard A. Fishel

Organizations

  • Thomas Jefferson University

Tags

DTIC Thesaurus Topics

  • Breast Cancer
  • Cancer
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Chromosome Aberrations
  • Chromosomes
  • Deoxyribonucleic Acids
  • Genetic Structures
  • Genetics
  • Genomic Instability
  • Health Services
  • Metabolic Diseases
  • Multiprotein Complexes
  • Neoplasms
  • Proteins
  • Skin Diseases

Fields of Study

  • Biology

Readers

  • Molecular Genetics
  • Molecular and genetic basis of cancer.
  • Systems Analysis and Design

Technology Areas

  • Biotechnology