Growth Factor Antagonism in Breast Cancer Chemotherapy

Abstract

The primary focus of this project is the first step in the aberrant cell signaling pathways that lead to uncontrolled proliferation and cancer, namely the interaction of growth factors with their receptor tyrosine kinases (RTKs). Our principal goal is to design growth factor antagonists that can inhibit ligand-induced receptor activation as a route to novel anti-cancer drugs. During the past year we have made important progress is establishing the goals of this project. We have developed efficient and direct synthetic routes to protein binding agents. We have extended our approach with the synthesis of some tetraphenylporphyrin based receptors that bind to proteins with high affinity. Most importantly, we have demonstrated that a member of our first generation of protein binding agents is able to bind to the surface of platelet derived growth factor. This binding is strong (IC50 < 250nM) under physiological conditions and is sufficient to block the activation of PDGF receptor tyrosine kinase. These results represent important steps is establishing the novel approach to breast cancer treatment that is the foundation of this project.

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Document Details

Document Type
Technical Report
Publication Date
May 01, 2000
Accession Number
ADA385735

Entities

People

  • A. Hamilton

Organizations

  • Yale University

Tags

DTIC Thesaurus Topics

  • Amino Acids
  • Breast Cancer
  • Cell Physiological Processes
  • Chemical Compounds
  • Chemical Synthesis
  • Chemistry
  • Demographic Cohorts
  • Growth Factors
  • Macrocyclic Compounds
  • Materials
  • Molecules
  • Neoplasms
  • Organic Chemistry
  • Porphyrins
  • Programmed Cell Death
  • Recognition
  • Tyrosine

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Oncology (Cancer Research).