Mitosis-Specific Negative Regulation of EGF-receptor in Breast Cancer: Molecular Mechanisms, Biological Significance and Therapeutic Application

Abstract

This project is to study the mechanisms of regulation of the EGFR during the cell cycle, in breast epithelial cells. Especially how cell which overexpress EGFR can overcome the negative regulation. We plan to use this negative regulation of the EGFR during the M phase of the cell cycle to develop a novel therapeutic strategy that uses the combination of EGF-PE and taxol as treatment. In trying to elucidate the mechanism of action of EGFR we have shown that cdc2 which can physically associate with neu, cannot associate with EGFR. Also we have shown that in vitro with treatment of EGF-PE and taxol together we do see that EGFR overexpressing cells are selectively killed, i.e. normal cells are protected from the EGF-PE by taxol treatment This novel approach may be a potential therapeutic strategy to treat breast cancer patients in the future. In addition to our proposed work we have discovered that cyclin Dl which is unregulated by EGFR can also be up regulated by beta-catenin. This work while not directly related to this proposal does help in understanding EGFR signaling pathways. Also this is a very novel finding of the correlation of the unregulation of beta-catenin and breast cancer.

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Document Details

Document Type
Technical Report
Publication Date
May 01, 2000
Accession Number
ADA387089

Entities

People

  • Stephanie M. Miller

Organizations

  • University of Texas at Austin

Tags

DTIC Thesaurus Topics

  • Antibodies
  • Biological Factors
  • Biomedical And Dental Materials
  • Biomedical Research
  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Molecular Biology
  • Neoplasms
  • Oncology
  • Peptides
  • Proteins

Fields of Study

  • Biology
  • Medicine

Readers

  • Military Logistics and Supply Chain Management
  • Molecular and Cellular Biology
  • Prostate Cancer Biology.