Role of Accessory Molecular in Endotoxin-Endothellial Interactions and Endothelial Barrier Dysfunction
Abstract
Bacterial lipopolysaccharide (LPS) is a component of Gram-negative bacteria endothelial barrier function. We used an in vitro experimental system in which LPS derived from Escherichia coli 0111:B4 was presented to postconfluent bovine pulmonary artery endothelial cell (EC) monolayers cultured on filter supports mounted in chemotaxis chambers. Transendothelial flux of 14C-bovine serum albumin was used as a quantitative measurement of paracellular permeability. We studied the roles of protein tyrosine phosphorylation and apoptosis in LPS-induced opening of the paracellular pathway and established that the lipid A moiety was the active portion of the LPS molecule in the non CD14-bearing EC system.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jun 01, 1998
- Accession Number
- ADA388219
Entities
People
- Simeon E. Goldblum
Organizations
- University of Maryland School of Medicine