Estrogens, Microtubules and Aneuploidy: Mechanisms of Mammary Gland Tumorigenesis
Abstract
This project assesses possible mechanisms of tumorigenesis for estradiol and related estrogens. The intent is to use mammary epithelial cells (MEC) from rat strains which differ markedly in their susceptibility to estrogen carcinogenicity. The hypothesis to be tested is that MEC from the sensitive strain will show increased sensitivity to microtubule disruption and the induction of aneuploidy by estrogens than will the cells from the resistant strain. This hypothesis has not been tested in the past year, and the author has tried repeatedly to obtain authorization for a new year no-cost extension of the project.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 2001
- Accession Number
- ADA396400
Entities
People
- Gregory A. Reed
Organizations
- University of Kansas Medical Center