Genes Controlling Susceptibility to Mammary Cancer
Abstract
This work examined the response of different genotypes of mice to the carcinogen 7,l2-dimethylbenzAanthracene (DMBA). Mice demonstrate a genotypic specificity for the particular tumors that they develop; DMPA increase the incidence of and decreases the average age at which these tumors are manifest. It does not appear that the genetic instability of the sort associated with the Werner syndrome in humans appreciably enhances the predilection of mice to develop mammary cancer, even in mice dosed with the carcinogen DMBA. Increased adiposity is a risk factor for postmenopausal breast cancer in humans. Data is presented demonstrating that the dietary intervention of calorie restriction reduces the incidence of mammary tumors following carcinogen administration in genotypes predisposed to its development.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 2001
- Accession Number
- ADA396452
Entities
People
- Ruth Lipman
Organizations
- Tufts University