Cell Cycle Regulation by TGFb Signaling in C. elegans

Abstract

One of the most potent inhibitors of epithelial growth is TGF beta. Understanding the connection between TGF beta and cell cycle control is an important avenue of experimentation towards treating breast cancers. We are utilizing two complementary approaches in C elegans to find cell cycle regulatory genes that respond to TGF beta. First, we are using a cell cycle reporter, ribonucleotide reductase, to monitor cell cycle activation in mutagenized animals that are arrested at the dauer stage (a TGF beta induced developmental stage). Appropriate genetic strains have been constructed and tested, which will allow screening to proceed shortly. In a second approach, we are probing DNA microarrays with mRNAs collected from animals entering dauer. This experiment should identify genes whose regulation is altered by TGF beta as the animals undergo cell cycle arrest. During the next year, we hope to have isolated mutants from our screen and to have begun the molecular characterization of them.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2001
Accession Number
ADA398212

Entities

People

  • Richard W. Padgett

Organizations

  • Rutgers University–New Brunswick

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Animals
  • Biomedical Research
  • Breast Cancer
  • Cell Physiological Processes
  • Cells
  • Dna Microarrays
  • Epithelial Cells
  • Genes
  • Genetics
  • Neoplasms
  • New Jersey
  • Nucleotides
  • Regulations
  • Worms

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Molecular Genetics

Technology Areas

  • Biotechnology