Mucin (MUC1) Expression and Function in Prostate Cancer Cells

Abstract

Mucin glycoproteins are highly expressed by many tumors, reduce normal cell-cell and cell-extracellular matrix adhesion and protect cancer cells from attack by the immune system. Mucin expression not only increases, but also changes from a restricted pattern of apical expression to a general distribution over the entire cell surface. In this regard, conversion of prostate epithelium from a highly-organized, growth-controlled phenotype to a highly proliferative, metastatic phenotype is associated with loss of cell polarity. Very few studies been performed on mucin expression by prostate cancer cells. MUC1 is a large molecular weight, type I transmembrane mucin glycoprotein expressed by normal and malignant prostate epithelium. High level cell surface expression, reported immunosupressive activities of its released ectodomain, and antiadhesive properties all contribute to this mucin's ability to protect and promote tumor cell growth and survival. Recent observations using human breast cancer cell lines indicate that MUC1 can associate with the intracellular signal transducing molecules, beta-catenin and ORB-2. Recent studies from the Pi's lab demonstrate that cytokines, including interferon-gamma, markedly stimulate MUC1 gene expression. Primary prostate tumors are often found in the vicinity of cytokine producing cells, and commonly metastasize to bone arrow, a rich source of these same cytokines.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2001
Accession Number
ADA404636

Entities

People

  • Daniel D. Carson

Organizations

  • University of Delaware

Tags

DTIC Thesaurus Topics

  • Biochemistry
  • Biological Sciences
  • Biology
  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Cytokines
  • Glycoconjugates
  • Glycoproteins
  • Neoplasms
  • Polysaccharides
  • Prostate Cancer
  • Proteins
  • Tissues

Fields of Study

  • Biology
  • Medicine

Readers

  • Immunology
  • Molecular Biology and Genetics