Role of KIP2 in Breast Cancer

Abstract

The cell cycle is regulated by the action of a family of cyclin dependent kinases (Cdks) which catalyze particular cell cycle transitions. Cdks are positively regulated through interaction with cyclins and are negatively regulated through phosphorylation and through association with inhibitory proteins of the CIP/KIP and INK4 families. Our research has focused on the role of p57KIP2 in development and cancer. We have found that p57KIP2 is expressed in a highly cell type specific manner during embryonic development and in adult tissues being most highly expressed in terminally differentiated cells. Through analysis of p57K1P2 deficient mice, however, we have found that p57K1P2 is required for normal development of several tissues including kidney, lends, muscle, and bone. We also discovered the p57 is involved in the human overgrowth and cancer predisposition disease Beckwith-Weidemann Syndrome, BWS. Current studies are aimed at understanding in greater detail the function of p57KIP2 and its possible role in human cancers.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2001
Accession Number
ADA405252

Entities

People

  • Stephen Elledge

Organizations

  • Baylor College of Medicine

Tags

DTIC Thesaurus Topics

  • Abdomen
  • Animal Structures
  • Breast Cancer
  • Carcinoma
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Chromosomes
  • Congenital Hereditary And Neonatal Diseases And Abnormalities
  • Diseases And Disorders
  • Embryos
  • Endocrine Glands
  • Fetus
  • Genetic Structures
  • Genetics
  • Neoplasms
  • Skeletal Muscle

Fields of Study

  • Biology

Readers

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