Isolation of Estrogen-Responsive Genes in Human Breast Cancer Cells

Abstract

The purpose of this proposal is to isolate and identify estrogen-responsive genes in human breast cancer cells using the chromatin immunoprecipitation (ChIP) protocol. Estrogen receptor (ER) -bound DNA has been isolated using ChIP from human breast cancer NCF-7 (T5) cells (ER positive and hormone dependent). Southern blotting analysis shows that ER-DNA contains ER-responsive sequences (ER, PR, P52 and c-myc). PCR analysis demonstrated that ER-DNA contains the p52 gene promoter, which is expressed in ER positive cells. Recent evidence shows that ER and associated coactivators are recruited to the estrogen inducible promoters in a cyclical manner. It is reasonable to believe that ER interaction with DNA is dynamically altered during estrogen induction. ER-bound DNA from MCF-7 (T5) cells treated with estradiol (E2) for 30, 60 and 120 min were isolated and analyzed by POR. A library of ER-bound DNA from 30 min E2 treated MCF-7 (T5) cells has been constructed. ChIP-ER-DNA fragments were cloned, sequenced, and blast searched in GenBank. Approximately 900 positive colonies were collected from this library, and approximately 100 cloned DNA fragments were sequenced. A - 1/2(ERE)-Sp1 sequence has been identified in PACE4 (pro-protein convertase) and EWS (Ewing's sarcoma) genes. Studies to determine whether these genes respond to estrogen is currently being determined.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 2002
Accession Number
ADA411259

Entities

People

  • James R. Davie

Organizations

  • University of Manitoba

Tags

DTIC Thesaurus Topics

  • Antineoplastic Agents
  • Birds
  • Breast Cancer
  • Carrier Proteins
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Colon Cancer
  • Enzyme Inhibitors
  • Epithelial Cells
  • Estrogens
  • Genetic Structures
  • Hormones
  • Neoplasms
  • Proteins
  • Tumor Cell Line

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Molecular Genetics