Gene Methylation in Prostate Cancer

Abstract

Background: NKX3. 1 is a homeoprotein with prostate-specific expression in adults. Loss of NKX3.1 correlates with prostate cancer progression. Loss of heterozygosity affects NKK3.1 in about 80% of prostate cancers. This project focuses on DNA methylation of the NKK3.1 gene promoter in prostate cancer cell lines and tissues. Materials and methods: DNA was analyzed by methylation-specific PCR and sequencing of bisulfite-treated DNA. We also treated cultured cells with the methylation ihhibitor 5-azacytidine and the histone deacetylase inhibitor, trichostatin A. Results: There was no effect of 5-azacytidine or trichostatin A on NKY3.1 expressionin cultured cells. By bisulfite modified DNA sequencing, we identified some methylated or partial methylated CpU islands in -1056 to 1172 of NKX3.1 gene. Some of tbem are selected to study their methylated situation in human prostate cancer tissues. We also study the effect of Sp transcription factor family on the expression of NKX.3.l, our results showed Spl and 5p3 did not influence on the expression of NKX3. 1. Conclusion: Some CpG islands in NKX3.l gene are methylated or partial methylated.

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Document Details

Document Type
Technical Report
Publication Date
Jan 01, 2003
Accession Number
ADA416621

Entities

People

  • Edward P. Gelmann
  • Huang Wen-xin

Organizations

  • American University

Tags

DTIC Thesaurus Topics

  • Abstracts
  • Alkanes
  • Androgen Receptors
  • Cell Line
  • Cells
  • Cultured Cells
  • Dna Sequence Analysis
  • Gene Expression
  • Inhibitors
  • Methylation
  • Neoplasms
  • Prostate
  • Prostate Cancer
  • Proteins
  • Tissues
  • Transcription Factors

Fields of Study

  • Biology

Readers

  • Molecular and genetic basis of cancer.