Transcription-Coupled Repair and Breast Cancer

Abstract

The overall goals of the project were to investigate the consequences of mutations in BRCA1 and BRCA2 in transcription-coupled repair (TCR) of oxidative damage and ultraviolet light induced damage in mouse and human systems. Since others (Gowan et al, 1998) have focused on mouse cell lines, we have focused on human cell lines. We examined the removal of UV light induced cyclobutane pyrimidine dimers (CPDs) from each strand of the DHFR gene in a BRCA1 mutant human cell line HCC1937. We find no evidence for a defect in TCR. We find no evidence for a defect in TCR in a BRCA2 deficient human cell line, Capan-1. Hence, defects in BRCA1 and BRCA2 do not appear to influence transcription-coupled repair of UV damage. The nucleotide excision repair pathway removes UV light-induced damage and bulky adducts formed in DNA by certain carcinogens.

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Document Details

Document Type
Technical Report
Publication Date
Jun 01, 2002
Accession Number
ADA417977

Entities

People

  • Isabel Mellon

Organizations

  • University of Kentucky

Tags

DTIC Thesaurus Topics

  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Diseases And Disorders
  • Escherichia Coli
  • Excision
  • Free Radicals
  • Genetics
  • Indicator Dyes
  • Ionizing Radiation
  • Neoplasms
  • Organic Chemistry
  • Skin Cancer
  • Testicular Cancer

Fields of Study

  • Biology

Readers

  • Molecular Genetics
  • Molecular and Cellular Biology
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