Application of Proteomics to Elucidate the Mechanism of Toxicity of the Chemical Warfare Agent Sulfur Mustard

Abstract

The complex molecular and cellular changes that occur following exposure to sulfur mustard (SM) have made it difficult to elucidate the primary mechanisms associated with SM-induced skin vesication. This report describes the use of proteomic approaches and methodologies to address this problem. Proteomic technologies, primarily two-dimensional (2D) gel electrophoresis and mass spectrometry, allow monitoring and identification of the complex molecular changes that occur in cells following toxic exposures. Cultured human epidermal keratinocytes were exposed to a vesicating dose (200 mM) of SM for up to 24 hours, the cellular proteins were extracted, and were analyzed by 2D electrophoresis. Selected differentially expressed proteins were analyzed by mass spectrometry for identification. Using this approach we have identified proteins involved in cytoskeletal structure as potential targets of SM. We are applying these techniques to identify protein pathways involved in the molecular mechanism of SM-induced vesication, and to identify potential therapeutic targets for the development of vesicant medical countermeasures.

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Document Details

Document Type
Technical Report
Publication Date
May 01, 2003
Accession Number
ADA418243

Entities

People

  • James F. Dillman
  • John J. Schlager

Organizations

  • United States Army Medical Research Institute of Chemical Defense

Tags

DTIC Thesaurus Topics

  • Biotechnology
  • Cells
  • Chemical Warfare
  • Chemical Warfare Agents
  • Chemistry
  • Electrophoresis
  • Epithelial Cells
  • Gel Electrophoresis
  • Identification
  • Mass Spectra
  • Mass Spectrometry
  • Peptides
  • Proteins
  • Proteomics
  • Spectrometry
  • Toxicity
  • Two Dimensional

Readers

  • Geochemistry
  • Molecular Genetics

Technology Areas

  • Biotechnology