Cellular Retinoic Acid Binding Protein and Breast Cancer

Abstract

In attempts to understand how the signaling by retinoic acid (the active vitamin A metabolite) is regulated we have been studying the retinoic acid binding protein called CRABP-II. These studies revealed that CRABP-II acts to enhance the transcriptional activities of RA and that it does so by directly delivering the hormone to its cognate transcription factor, RAR. Consequently CRABP-II dramatically sensitized cultured mammary carcinoma cells to RA-induced growth inhibition. Similarly, over-expression of CRABP-II inhibited mammary tumor growth in two different mouse models of cancer. CRABP-II may be a novel target for therapeutic and preventive strategies for retinoid-treatment of breast cancer. This project aims to delineate the mechanism by which CRABP-II modulates RA activity, especially as related to its ability to enhance the anti-proliferative action of the ligand. The first aim is to determine the extent to which CRABP-II acts in activating different isotypes of RAR. The second aim is to dissect the mechanism by which RA-induced growth inhibition is mediated. This will then allow for closer inspection of particular target genes that are under such control. The third aim is to understand the basis for RA-resistance of mammary carcinoma cells and how CRABP-II functions to overcome this resistance.

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Document Details

Document Type
Technical Report
Publication Date
May 01, 2004
Accession Number
ADA425991

Entities

People

  • Leslie J. Willmert

Organizations

  • Cornell University

Tags

DTIC Thesaurus Topics

  • Acids
  • Apoptosis
  • Breast Cancer
  • Carrier Proteins
  • Cell Physiological Processes
  • Cells
  • Chemical Compounds
  • Chemical Synthesis
  • Chemistry
  • Inhibition
  • Neoplasms
  • Programmed Cell Death
  • Proteins
  • Resistance
  • Retinoic Acids
  • Tumor Cell Line

Fields of Study

  • Biology
  • Chemistry

Readers

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