AKT2 Oncogene and Phosphoinositide 3-Kinase in Human Breast Cancer

Abstract

By screening NCI diversity set that was derived from 140,000 compounds, we have identified several potential Akt inhibitors, one of which has been further characterized and named Akt/PKB inhibitor (API)-2. API-2 specifically inhibited kinase activity of AKT1, AKT2 and AKT3. The inhibition of Akt kinase resulted in suppression of cell growth and induction of apoptosis in human cancer cells that harbor constitutively activated Akt due to overexpression of Akt or other genetic alterations such as PTEN mutation. Furthermore, API-2 potently inhibited tumor growth in nude mice of human cancer cells where Akt is aberrantly expressed/activated but not of those cancer cells where it is not. In addition, we have demonstrated Akt phosphorylation of XIAP to exert its cell survival signal at postmitochondrial level. We have also shown that Akt is targeted by geranylgeranyltransferase I inhibitors (GGTIs).

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2004
Accession Number
ADA430371

Entities

People

  • Jin Q. Cheng

Organizations

  • University of South Florida

Tags

DTIC Thesaurus Topics

  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Health Services
  • Medical Personnel
  • Neoplasms
  • Oncology
  • Programmed Cell Death

Fields of Study

  • Biology
  • Chemistry

Readers

  • Molecular Biology and Genetics

Technology Areas

  • Biotechnology