Humanizing the Mouse Androgen Receptor to Study Polymorphisms and Mutations in Prostate Cancer
Abstract
Androgen receptor (AR) is a critical component in prostrate oncogenesis. AR alleles varying in length of the Nterminal glutamine (Q) tract carry distinct disease risks. Mutation of AR in tumors may influence androgen independence and resistance to treatment. Difficulties studying cancer initiation and progression are partly overcome with transgenic mouse tumor models. However, the mouse AR N-terminus differs significantly from human.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jan 01, 2005
- Accession Number
- ADA434360
Entities
People
- Diane Robins
Organizations
- University of Michigan