MIC-1, A Potential Inhibitor of Breast Tumor Progression

Abstract

MIC-l is a member of the transforming growth factor-beta family of growth and differentiation factors. MIC-l is expressed in breast tissue and has numerous specific effects including inhibiting both breast development and breast duct proliferation. We hypothesize, therefore, that MIC-l may play a role in inhibiting breast cancer progression. In order to study this question we have generated genetically modified mi in which murine MIC-l expression is abolished (null mice). We are in the process of breeding these MIC-1 null mice with mouse lines that are predisposed to the development breast cancer and this will be examined in the upcoming year as a no-cost extension of this grant. We will analyze the effects of the absence of MIC-l on the rate of breast malignancy development in these animals comparing it to those that have intact MIC-l production. If our hypothesis is correct, MIC-l null mice will exhibit increased incide and/or severity of breast cancer. As such, these studies may identify an important anti tumor pathway in breast cancer, potentially providing novel strategies and targets for chemical therapeutics or diagnosis.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 2004
Accession Number
ADA435049

Entities

People

  • Leonidas G. Koniaris

Organizations

  • University of Miami

Tags

DTIC Thesaurus Topics

  • Abstracts
  • Biomedical Research
  • Breast Cancer
  • Breeding
  • Cancer
  • Genotypes
  • Growth Factors
  • Inhibitors
  • Neoplasms
  • Peptide Growth Factors
  • Peptides
  • Production
  • Therapy

Fields of Study

  • Biology

Readers

  • Criminal Law
  • Oncology (Cancer Research).

Technology Areas

  • Biotechnology