An Epithelial-Derived, Integral Membrane, Kunitz-Type serine Protease Inhibitor in Breast Cancer

Abstract

In the current research plan, we proposed to study the anti-tumor and anti-protease activity of a membrane-bound Kunitz-type serine protease inhibitor (KSPI; also known as HAI-1). In order to investigate how HAI-1 regulates matriptase function, we investigated the cellular events associated with matriptase activation. During matriptase activation induced either by S1P or suramin, HAI-1 along with matriptase is translocated and accumulated at cell-cell junctions or in vesicle-like structures, which were named as matriptase activation foci. In activation foci, HAI-1 binds active matriptase to form a 120-kDa complex immediately following the activation of matriptase. The close temporal and spatial coupling of matriptase activation with its inhibition suggests that the proteolytic activity of this enzyme must be well controlled, and that the activation of matriptase substrates may be tightly regulated by this mechanism.

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Document Details

Document Type
Technical Report
Publication Date
Aug 01, 2004
Accession Number
ADA435266

Entities

People

  • Chen-yong Lin

Organizations

  • Georgetown University

Tags

DTIC Thesaurus Topics

  • Abstracts
  • Biomedical Research
  • Breast Cancer
  • Cancer
  • Cell Shape
  • Cells
  • Cellular Structures
  • Couplings
  • Enzyme Inhibitors
  • Enzymes
  • Epithelial Cells
  • Growth Factors
  • Inhibition
  • Inhibitors
  • Integrals
  • Intercellular Junctions
  • Neoplasms

Fields of Study

  • Biology

Readers

  • Cellular and Molecular Pathways of Apoptosis.
  • Molecular and Cellular Biochemistry