Paracrine Regulation of Prostatic Carcinogenesis
Abstract
This report summarizes the work performed during this study. The long term goal of this project is to better understand why some prostate tumors grow aggressively while others are extremely slow growing lesions. The objective of the proposed research is to establish immortalized stromal cell lines denved from normal human prostate and from human prostate cancer and to use these cells to investigate the role of IGFs in prostate cancer growth. A number of new molecular tools were generated in the course of this project. A new technology (RNA interference) came into common use allowing us to suppress gene expression in a manner of technical complexity to overexpression of genes. We have been one of the first groups to be able to use this approach in tissue recombination models. As a result we have been able to identify the effects of suppression of IGF signaling in human prostate cancer cells in vivo as a mechanism to inhibit invasion and enhance differentiation.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jan 01, 2005
- Accession Number
- ADA435853
Entities
People
- Simon W. Hayward
Organizations
- Vanderbilt University Medical Center