Anti-Estrogen Regulation of Macrophage Products That Influence Breast Cancer Cell Proliferation and Susceptibility to Apoptosis

Abstract

Macrophage function can be regulated by breast cancer cells to sustain an inflammatory microenvironment that promotes cancer cell proliferation and survival. MCF-7 cells suppress IL-10 expression by co-cultured macrophages. Activated and resting THP-1 macrophages were able to induce SNAIL expression in MCF-7 cells. SNAIL has been implicated in the epithelial-mesenchymal transition which precedes metastasis. These observations suggest that tumor-associated macrophages may promote progression-related gene expression at discrete steps in the progression cascade. Both Faslodex(TradeMark) and tamoxifen were effective in up-regulating pigment epithelium-derived factor, a potent anti-angiogenic cytokine, and in down-regulating SNAIL in MCF-7 cells.

Open PDF

Document Details

Document Type
Technical Report
Publication Date
Aug 01, 2005
Accession Number
ADA442860

Entities

People

  • Theodore A. Bremner

Organizations

  • Howard University

Tags

DTIC Thesaurus Topics

  • Apoptosis
  • Biological Sciences
  • Breast Cancer
  • Cell Biology
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Culture Techniques
  • Genetics
  • Medical Personnel
  • Molecular Biology
  • Neoplasms
  • Oncology
  • Proteins
  • Students
  • Tumor Cell Line

Fields of Study

  • Biology
  • Medicine

Readers

  • Breast cancer cell signaling and growth regulation.
  • Immunology
  • Molecular Biology and Genetics