Regulation of Intracellular pH in Lungs and Other Tissues During Hypercapnia

Abstract

Using a 14C-labeled DMO, 36Cl, and 3H method, we have determined the in vivo buffering capacity of lung, kidney, heart, skeletal muscle, and extracellular fluid (ECF) of guinea pigs during hvpercapnia. After 1 day's exposure to 15% CO2, both the relative CO2 buffer values and the "%pH regulation" were lung > kidney > heart > ECF > skeletal muscle. For lung tissue the intracellular pH was significantly decreased only during acute (8 h) hypercapnia and had completely returned to control values after 7 days with arterial Pco-2 = 122 Torr. Kidney and cardiac muscle also showed ca. 100% regulation of pH at 7 days, whereas skeletal muscle and ECF showed only 80 and 70% pH regulation, respectively. The results are discussed with respect to the important (and pH-dependent) metabolic functions of the lung and kidney.

Open PDF

Document Details

Document Type
Technical Report
Publication Date
Mar 10, 1978
Accession Number
ADA446584

Entities

People

  • Karl E. Schaefer
  • Stephen C. Wood

Organizations

  • Naval Submarine Medical Research Laboratory

Tags

DTIC Thesaurus Topics

  • Acid-Base Imbalance
  • Acids
  • Animals
  • Blood
  • Cardiovascular Diseases
  • Cells
  • Cellular Structures
  • Diseases And Disorders
  • Fluids
  • Heart
  • Membrane Potentials
  • Metabolism
  • Physiology
  • Respiration
  • Rodents
  • Skeletal Muscle
  • Tissues

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Toxicology/Environmental Toxicology
  • Trauma Surgery or Emergency Medicine.