Analysis of Morphogenic Effect of hDAB2IP on Prostate Cancer and its Disease
Abstract
Prostate homeostasis is a balance between cell proliferation, apoptosis and differentiation in basal and luminal epithelia. It is believed that loss of homeostatic control in these cells renders the onset of neoplasm in prostatic epithelium. Until now, a little is known about homeostatic machinery in normal prostatic epithelium. In this project, we have proposed that DAB2IP protein, a novel RASGAP, is a part of homeostatic machinery and plays an important in modulating signal pathways elicited by exogenous growth/death stimuli. For example, DAB2IP is able to counteract androgen-mediated cell growth and is involved in TNF-alpha-mediated cell apoptosis. We further demonstrated that decreased steady-state levels of DAB2IP in normal prostatic epithelium resulted in the tumor formation of these cells. Further investigation has led us to unveil the underlying mechanism of DAB2IP in modulating specific signal cascades.
Document Details
- Document Type
- Technical Report
- Publication Date
- Feb 01, 2006
- Accession Number
- ADA452524
Entities
People
- Jer-Tsong Hsieh
Organizations
- University of Texas at Dallas