Regulation of MDM2 Activity by Nucleolin

Abstract

A key antagonist of the p53 tumor suppressor is human MDM2 (Hdm2). We examined the significance of a recently identified complex between Hdm2 and nucleolin a c-Myc-induced gene product with defined roles in ribosomal RNA processing and the inhibition of chromosomal DNA replication following stress. Changes in the level of nucleolin protein in unstressed cells cause parallel changes in the amount of pS3 protein. Alterations in pS3 levels arise from nucleolin binding to the p53-antagonist Hdm2 resulting in the inhibition of both pS3 ubiquitination and Hdm2 auto-ubiquitination. Unexpectedly we find that nucleolin also reduces Hdm2 protein levels demonstrating that nucleolin inhibits Hdm2 using multiple mechanisms. Increases in nucleolin levels in unstressed cells led to higher expression of p2lcipl/wafl a reduced rate of cellular proliferation and an increase in apoptosis. Thus nucleolin has a number of properties in common with the tumor suppressor ARF. We propose that nucleolin like ARF responds to hyper-proliferative signals by up-regulation of pS3 through Hdm2 inhibition. These findings have important implications on the progression of breast cells to cancer and have the potential to provide new therapeutic routes to treat breast cancers.

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Document Details

Document Type
Technical Report
Publication Date
Jun 01, 2006
Accession Number
ADA457556

Entities

People

  • James A. Borowiec

Organizations

  • Grossman School of Medicine

Tags

Communities of Interest

  • Air Platforms
  • Biomedical

DTIC Thesaurus Topics

  • Apoptosis
  • Biomedical And Dental Materials
  • Biotechnology
  • Breast Cancer
  • Carrier Proteins
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Confocal Microscopy
  • Fungi
  • Neoplasms
  • Organelles
  • Polymeric Films
  • Proteins
  • Proteomics
  • Tumor Cell Line

Fields of Study

  • Biology

Readers

  • Cellular and Molecular Pathways of Apoptosis.
  • Optical Fiber Sensing and Electromagnetic Propagation.