Bioenergetic Approaches and Inflammation of MPTP Toxicity
Abstract
We wish to continue to examine a number of new neuroprotective agents ill the MPTP model of PD, which act by inhibiting the mitochondrial permeability transition (MPT) We also wish to utilize metabolomic profiling to identify novel biomarkers for PD and to investigate whether these occur ill animal models of PD. We will develop and characterize a new animal model of PD making a knockout of PINK 1, which is a nuclear-encoded kinase localized to mitochondria, and which causes autosomal recessive PD. Lastly, we wish to study the effects of human dopaminergic stem cells in the 6-hydroxy- dopamine model of PD.
Document Details
- Document Type
- Technical Report
- Publication Date
- Sep 01, 2006
- Accession Number
- ADA462190
Entities
People
- M. F. Beal
Organizations
- Cornell University