Targeting Mechanisms of Resistance to Taxane-Based Chemotherapy

Abstract

Patients with high-risk localized prostate cancer have a high recurrence rate following primary therapy. Neoadjuvant chemotherapy has been shown to be beneficial in reducing recurrence rates in some tumor types, but has yet to be of proven benefit in prostate cancer. Further, current clinical, pathological and molecular markers poorly predict the response and resistance of chemotherapy, and the molecular mechanisms of chemotherapy resistance are largely unknown. We utilized tissue resources from a unique prospective phase II clinical trial of neoadjuvant chemotherapy with docetaxel and mitoxantrone in patients with high-risk localized prostate cancer to identify molecular alterations after chemotherapy, and correlated these alterations with clinical and pathological indicators of tumor response. We hypothesized that this approach may identify molecular signatures of chemotherapy resistance and uncover mechanisms or pathways suitable for targeting with the objective of improving tumor responses to chemotherapy. Gene expression changes after chemotherapy were measured in 31 patients who completed 4 cycles of docetaxel and mitoxantrone neoadjuvant chemotherapy. After excluding possible ischemia-related genes, the expression of 53 genes were significantly altered after chemotherapy. Several cytokines were significantly up-regulated including IL-8, CCL2, GDF15, CXCL10, and IL1B. Overexpression of GDF15 or treatment with GDF15 protein in DU145 cells conferred resistance to docetaxel and mitoxantrone. Using PSA decline greater than 40% as a cut-point to distinguish good from poor responders, we were able to identified 33 significantly-altered genes. IL8 was not only shown to be activated after chemotherapy but also have higher expression levels in the group of poor responders compared with good responders. Alterations of molecular signatures after administration of docetaxel and mitoxantrone in patients with high-risk localized prostate cancer were recognized.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2006
Accession Number
ADA462817

Entities

People

  • Chung-yung Huang

Organizations

  • Fred Hutchinson Cancer Center

Tags

DTIC Thesaurus Topics

  • Biological Processes
  • Biomedical Research
  • Biopsy
  • Blood
  • Cancer
  • Cell Physiological Processes
  • Cells
  • Clinical Trials
  • Culture Techniques
  • Diseases And Disorders
  • Gene Expression
  • Neoplasms
  • Prostate
  • Prostate Cancer
  • Proteins
  • Therapy
  • Tissues

Fields of Study

  • Medicine

Readers

  • Molecular and genetic basis of cancer.
  • Prostate Cancer Biology.