Homeostatic T Cell Expansion to Induce Anti-Tumor Autoimmunity in Breast Cancer

Abstract

We have previously shown that effective anti-tumor autoimmunity can be induced if a tumor-cell challenge is given to mice undergoing homeostatic T-cell proliferation, a process dependent on signaling by self-peptide/MHC and trophic cytokines. We investigated whether this principle can be applied to mouse models of advanced breast carcinoma, and whether the anti-tumor response can be enhanced using selected T-cell subpopulations, cytokines and tumorvaccines. The results indicated that (a) homeostatic T-cell proliferation consistently elicits anti-tumor responses; (b) irradiation is more effective than Tcell depletion by antibodies in inducing anti-tumor responses mediated by homeostatic T-cell proliferation; however, irradiation (and/or the resulting lymphopenic state) may facilitate metastasis dissemination; (c) the frequency of T regulatory (Treg) cells increases during homeostatic proliferation, particularly in the presence of a growing tumor; in vivo depletion of Treg cells enhances the anti-tumor effect of homeostatic T-cell proliferation on subcutaneous breast carcinoma; (d) gamma/delta T cells, a lymphocyte subpopulation with significant anti-tumor activity, can be induced to undergo homeostatic proliferation, and this requires depletion of both alpha/beta and gamma/delta T cell compartments and availability of either IL-7 or IL-15; (e) the anti-tumor response is diminished in aged mice, and this correlates with inefficient homeostatic T-cell proliferation; this defect can however be corrected by provision of the trophic cytokine IL-7; (f) IL-7 complexed with anti-IL-7 antibodies and/or IL-2 complexed with anti-IL-2 antibodies induce T cell proliferation in both lymphopenic and non-lymphopenic mice; (g) in non-lymphopenic mice, IL-7/antibody and particularly IL-2/antibody complexes induce cytotoxic effector functions in CD8 T cells, and inhibit tumor growth, metastasis and mortality in a model of breast carcinoma;

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Document Details

Document Type
Technical Report
Publication Date
Apr 01, 2007
Accession Number
ADA472124

Entities

People

  • Roberto Baccala

Organizations

  • Scripps Research

Tags

DTIC Thesaurus Topics

  • Antigen-Presenting Cells
  • Antigens
  • Apoptosis
  • Autoimmunity
  • Biological Factors
  • Biomedical Research
  • Breast Cancer
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Cytokines
  • Immune System
  • Lymphatic System
  • Lymphocytes
  • Neoplasms
  • Proteins
  • Tissues

Fields of Study

  • Biology
  • Medicine

Readers

  • Immunology
  • Oncology (Cancer Research).