Water Sparing in Chronic Ethanol Exposure is Associated With Elevated Renal Estrogen Receptor Beta and Vasopressin V2 Receptor mRNA in the Female Rate

Abstract

Fluid handling is known to differ between males and females. Interactions between sex steroids such as estrogen with fluid regulating hormones like vasopressin (VP) are likely key to establishing these differences. Research has identified estrogen receptor (ER) alpha and Beta in renal tissue which may affect renal fluid handling. Thus, this study hypothesized that chronic ethanol exposure would elicit different alterations to water load excretion between male and female Sprague Dawley (SD) rats due to changes in renal VP V2 receptor (V2R) or ER mRNA expression. Therefore, in this study we compared 120 minute excretion of a 2% Body Weight (BW) water load between male control (n=6) and ethanol-fed (n=14) rats, and female control (n=26) and ethanol-fed (n=26) rats. Additionally renal papilla mRNA expression of V2R, ERalpha, and ERbeta was compared between male control (n=S) and ethanol-fed (n=I 5) rats, and female control (n=12) and ethanol-fed (n=17) rats. Female ethanol fed rats showed a 16% reduction in water load excretion (p<O.05) compared to controls. RT-PCR analysis revealed that the decreased water excretion in ethanol-fed females was accompanied by a 40% increase in V2R mRNA (p<O.05) and a 146% increase in ERbeta mRNA (p<O.O5) in renal papilla tissue compared to controls. In contrast, ethanol treatment in male rats resulted in no difference in water excretion, and yielded no change to V2R or ERbeta mRNA expression in the renal papilla. ERbeta expression was not different between males and females, nor affected by ethanol treatment. Overall, these results suggest that females can better compensate for the dehydrating effects of ethanol exposure by increasing renal responsiveness to VP via upregulating renal V2R. Also, ethanol specifically upregulates the ER$ subtype in the female kidney which may modulate renal sensitivity to VP.

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Document Details

Document Type
Technical Report
Publication Date
Dec 01, 2007
Accession Number
ADA478071

Entities

People

  • Odaro J. Huckstep

Organizations

  • University of Hawaiʻi at Mānoa

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Air Force
  • Arteries
  • Blood
  • Blood Volume
  • Body Weight
  • Cardiovascular Physiological Phenomena
  • Cell Physiological Processes
  • Chemistry
  • Computer Programs
  • Department Of Defense
  • Electronic Mail
  • Estrous Cycle
  • Gene Expression
  • Hormones
  • Materials
  • Medical Personnel
  • National Security

Fields of Study

  • Medicine

Readers

  • Breast cancer cell signaling and growth regulation.
  • Cardiovascular Physiology
  • Toxicology/Environmental Toxicology