Estrogen-Induced Depurination of DNA: A Novel Target for Breast Cancer Prevention
Abstract
The research conducted in this COE grant is based on the paradigm that estrogens can become endogenous carcinogens when their metabolism is unbalanced, favoring formation of catechol estrogen quinones and their reaction with DNA. Compelling evidence obtained in the various specific aims of this COE will be decisive for determining the risk of breast cancer by using the depurinating estrogen-DNA adducts as biomarkers. These biomarkers will also be used for evaluating the ability of specific antioxidants to prevent breast cancer initiation. Much of the research accomplished by this COE was published in a review article in BBA-Reviews in Cancer, which was co-authored by all the participants of the COE.
Document Details
- Document Type
- Technical Report
- Publication Date
- May 01, 2008
- Accession Number
- ADA491583
Entities
People
- Ercole L. Cavalieri
Organizations
- University of Nebraska Medical Center