Beta 2-Microglobulin: A Novel Therapeutic Target for the Treatment of Human Prostate Cancer Bone Metastasis
Abstract
This proposal focuses on defining the role of beta 2-microglobulin (beta2-M) as a growth factor and signaling molecule contributing to prostate cancer bone metastasis. In this proposal, we proposed two Aims. Specific Aim 1 is to investigate the possible signaling pathway regulated by beta2-M in human prostate cancer cells with specific emphasis on the role of bradykinin (BK) receptor as the possible mediator of beta2-M signaling. We will define signaling transduction pathway mediated by beta2-M in promoting osteomimicry and prostate cancer cell growth and bone metastasis. Specific Aim 2 is to determine the effectiveness of BKM-1740, a BK receptor antagonist chemically conjugated with bisphosphonate known to accumulate in bone, in the inhibition of prostate cancer growth in mouse bone. By targeting BK receptor on the cell surface of prostate cancer cells using a synthetic BK inhibitor, BKM-1740, we expect this will increase prostate cancer cell death in bone.
Document Details
- Document Type
- Technical Report
- Publication Date
- Mar 14, 2009
- Accession Number
- ADA511994
Entities
People
- Leland W. Chung
Organizations
- Emory University