Bone Marrow Microenvironmental Control of Prostate Cancer Skeletal Localization
Abstract
The purpose of this study is to analyze the role of prostate cancer derived PTHrP and its ability to impact the bone marrow microenvironment of the skeletal metastasis. During the first year of the project, PC-3 cell lines expressing luciferase and with altered PTHrP levels were generated and their activity verified in in vitro and in vivo assays. The PC-3 cell lines with lowered PTHrP levels (via shRNA knock down) developed smaller tumors. The PC-3 cell lines with lowered PTHrP levels showed a trend toward lowered hematopoietic progenitors in the bone marrow. Altering the bone marrow microenvironment with an agent that reduces HPCs (cyclophosphamide) increased tumor engraftment. Altering the bone marrow microenvironment with zoledronic acid (a proven anti-tumor therapy) increased HPCs. These studies have already provided early data that suggest there are key and controllable events that can be triggered in the bone marrow which are conducive to tumor growth.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 2009
- Accession Number
- ADA514367
Entities
People
- Laurie K. Mccauley
Organizations
- University of Michigan