Differential Alterations in Excitatory and Inhibitory Networks Involving Dentate Granule Cells Following Chronic Treatment with Distinct Classes of NMDAR Antagonists in Hippocampal Slice Cultures

Abstract

Excessive N-methyl-D-aspartate receptor (NMDAR) activation is thought to contribute to brain diseases including epilepsy. However, we showed previously that electrographic seizures involving dentate granule cells induced by a GABAAR antagonist (BMI, 10 micrometers) or removal of Mg2+ (0 mM) were significantly increased following chronic D-APV treatment and modestly increased following chronic memantine treatment. Conversely BMI- and 0 mM Mg2+-induced seizures were dramatically reduced following chronic Ro25,6981 treatment. The goal of this study was to investigate potential mechanisms underlying the differential effects of distinct classes of NMDAR antagonists on seizures. To accomplish this goal, whole-cell electrophysiological recordings and morphological techniques were utilized to examine changes in synaptic connectivity in granule cells from hippocampal slice cultures treated with different classes of NMDAR antagonists.

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Document Details

Document Type
Technical Report
Publication Date
Mar 08, 2010
Accession Number
ADA539249

Entities

People

  • Shuijin He

Organizations

  • Uniformed Services University of the Health Sciences

Tags

DTIC Thesaurus Topics

  • Alzheimer Disease
  • Anticonvulsants
  • Brain
  • Cells
  • Chemistry
  • Epilepsy
  • Health Services
  • Neurodegeneration
  • Neurons
  • Neurosciences

Fields of Study

  • Biology

Readers

  • Neuroscience