Coordination of BRCA1/BARD1- and MRE11/RAD50/NBS1-Dependent DNA Transactions in Breast Tumor Suppression
Abstract
The main objective of the proposal is to understand how the Mre11-Rad50-Nbs1 complex and the BRCA1-BARD1 complex interact on DNA and coordinate DNA transactions that are critical for the maintenance of genomic stability and to prevent breast tumor development. We have started to characterize the behavior of BRAC1/BARD1 on DNA, using a single molecule approach. We have purified recombinant MRN and BRCA1/BARD1 complexes. We have shown that Whereas MRN is essential for processing DNA ends at double-strand breaks (DSBs), BRCA1 is dispensable for this function. In contrast both BRCA1 and MRN complex are required for checkpoint activation following DNA damage.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 2009
- Accession Number
- ADA548368
Entities
People
- Eric Greene
- Jean Gautier
Organizations
- Columbia University