Inhibition of Histone Deacetylases (HDACs) and mTOR Signaling: Novel Strategies Towards the Treatment of Prostate Cancer

Abstract

During the past year I have setup a transplant androgen sensitive and castrate resistant prostate tumor mouse model for pre- clinical evaluation of novel targeted therapies. Further, this tumor is derived from a transgenic mouse of prostate cancer and all studies are performed in immune-competent animals. We have currently submitted 2 manuscripts, the first describing the creation of the transplant tumor model, and the second describing the increased therapeutic efficacy of HDAC/mTOR inhibitor combination for the treatment of advanced and castrate resistant prostate cancer. We also describe that increased inhibition of androgen receptor and HIF-1 alpha signaling as a major reason for greater therapeutic efficacy by combination therapy. Lastly, this manuscript demonstrates that microRNA may provide novel biomarkers for indication of therapy response. This work is inline with a current clinical trial under recruitment to treat patients with HDAC/mTOR inhibitor combination who have castrate resistant prostate cancer.

Open PDF

Document Details

Document Type
Technical Report
Publication Date
Apr 01, 2012
Accession Number
ADA562460

Entities

People

  • Leigh Ellis

Organizations

  • Health Research, Incorporated

Tags

DTIC Thesaurus Topics

  • Androgen Receptors
  • Androgens
  • Biological Markers
  • Cell Count
  • Cell Line
  • Cell Physiological Processes
  • Clinical Trials
  • Combination Therapy
  • Diseases And Disorders
  • Inhibition
  • Inhibitors
  • Neoplasms
  • Prostate
  • Prostate Cancer
  • Proteins
  • Therapy
  • Tissues

Fields of Study

  • Biology
  • Medicine

Readers

  • Clinical Trial Research.
  • Molecular Biology and Genetics
  • Prostate Cancer Biology.

Technology Areas

  • Biotechnology
  • Biotechnology - Cancer Biotech