Role of NuSAP in Prostate Tumor Progression

Abstract

We have identified NuSAP as prognostic marker upregulated in recurrent prostate tumors. Our grant aimed at identifying the role of NuSAP in promoting proliferation and invasion in Prostate Cancer and identify genes that upregulate NuSAP expression. To characterize the role and regulation of NuSAP in prostate cancer, we studied the expression of NuSAP in the LNCaP and PC3 human prostate cancer cell lines. Post-transcriptional silencing of the NuSAP gene severely hampered the ability of PC3 to invade and proliferate in vitro. The promoter region of the NuSAP gene contains 2 CCAAT boxes and binding sites for E2F. Transient transfection of an E2F1 cDNA and 431 bp of the NuSAP promoter demonstrated E2F1 as an important regulator of expression. Deletion of the E2F binding site at nt -246 negated the effects of E2F1 on NuSAP expression. Electrophoretic mobility shift assays demonstrated that nuclear extracts of cells over-expressing E2F1 bound directly to the E2F binding site in the NuSAP promoter region. Finally, immunohistochemistry showed a strong correlation between E2F1 and NuSAP expression in human prostate cancer samples. NuSAP is a novel biomarker for prostate cancer recurrence after surgery and its over-expression appears to be driven in part by E2F1 activation.

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Document Details

Document Type
Technical Report
Publication Date
Jun 01, 2012
Accession Number
ADA562839

Entities

People

  • James D Brooks

Organizations

  • Stanford University

Tags

DTIC Thesaurus Topics

  • Androgen Receptors
  • Biological Markers
  • Biomedical Research
  • Breast Cancer
  • Cell Division
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Diseases And Disorders
  • Gene Expression
  • Institutional Review Board
  • Medical Personnel
  • Neoplasms
  • Prostate Cancer
  • Regulations
  • Regulators
  • Tissues

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Electrochemical Surface Science
  • Prostate Cancer Biology.