Maintenance of Epithelial Stem Cells by Cbl Proteins

Abstract

The Cbl family E3 ubiquitin ligases are critical regulators of tyrosine kinase-mediated signal transduction, and we and others demonstrated that the Cbl family proteins were required to maintain the homeostasis of the hematopoietic stem cell compartment and disturbance of this critical regulatory pathway led to cancer in human and mice. However, their roles in the epithelial stem/progenitor cells has never been examined. Using the newly-established hTERT-immortalized human mammary epithelial cell (hMEC) line (hereafter called TERT-hMEC), we demonstrated that loss of Cbl inhibited differentiation of epithelial cells. Our preliminary studies indicate that Cbl functions in multiple signaling pathways beyond the traditional EGFR pathway, which appears to be responsible for the complex phenotypes of Cbl deficiency in the epithelial compartment.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2012
Accession Number
ADA568740

Entities

People

  • Mayumi Naramura

Organizations

  • University of Nebraska Medical Center

Tags

DTIC Thesaurus Topics

  • Breast Cancer
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Epithelial Cells
  • Genetics
  • Intercellular Junctions
  • Lymphocytes
  • Peptide Growth Factors
  • Proteins
  • Stem Cells
  • Three Dimensional

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.

Technology Areas

  • Biotechnology