Development of a Combination Therapy for Prostate Cancer by Targeting Stat3 and HIF-1alpha

Abstract

It is becoming increasingly evident that improving the cure rate of many cancers will require treatment regimens hit more than one validated tumor targets. Developing an anti-cancer agent that targets two oncorproteins simultaneously is a promising strategy for accomplishing this goal. It would be expected to promote drug efficacy, reduce therapy-resistant without introducting additional toxic side effects. Recently, we have identified TEL03 from Chinese medicinal herb significantly inhibits activations of HIF-1alpha/2alpha and phosphorylated Stat3 (p-Stat3), and blocks the expression of their down-regulated oncogenes (e.g. Bcl2, VEGF, Glut1, and others) in cancer cells. TEL03 also dramatically suppressed the growth of prostate, breast and pancreatic tumors in xenograft models. Our previous studies provided evidence that targeting both HIF-1alpha and Stat3 together could improve tumor response to either agent alone, and reduce drug resistance and treatment failure.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2013
Accession Number
ADA580049

Entities

People

  • Naijie Jing

Organizations

  • Baylor College of Medicine

Tags

DTIC Thesaurus Topics

  • Alcohols
  • Amino Acids
  • Breast Cancer
  • Cancer
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Department Of Defense
  • Immune System
  • Molecular Dynamics
  • Neoplasms
  • Oncology
  • Prostate Cancer
  • Proteins
  • Surface Plasmon Resonance
  • Toxicity

Fields of Study

  • Medicine

Readers

  • Cellular and Molecular Pathways of Apoptosis.
  • Molecular Biology and Genetics
  • Oncology