Variations of Human DNA Polymerase Genes as Biomarkers of Prostate Cancer Progression

Abstract

Human DNA polymerases beta, eta, and kappa are enzymes that function in repairing damaged DNA. We set out to identify and characterize the role of prostate tumor mutations in human genes POLB, POLH and POLK. We discovered missense mutations in all three polymerase genes. Biochemical analysis of the polymerase beta missense variations showed that all of these somatic mutations affect either polymerase activity, enzyme stability, DNA synthesis fidelity, or a novel endonuclease activity. We further showed that the Pol beta endonuclease activity prevents aneuploidy and genomic instability. Thus, frequent polymerase mutations may contribute to prostate cancer progression, through distinct enzymatic functions. These mutations are valuable as biomarkers of cancer progression.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2013
Accession Number
ADA591964

Entities

People

  • Nick Makridakis

Organizations

  • Tulane University of Louisiana

Tags

DTIC Thesaurus Topics

  • Biological Markers
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Chromosome Aberrations
  • Chromosome Structures
  • Environmental Health
  • Genetic Structures
  • Genetics
  • Genomic Instability
  • Liquid Chromatography
  • Neoplasms
  • Prostate
  • Prostate Cancer
  • Reliability
  • Spreadsheet Software
  • Tissues

Fields of Study

  • Biology

Readers

  • Analytical Mechanics
  • Molecular Genetics
  • Molecular and genetic basis of cancer.