Overcoming Autophagy to Induce Apoptosis in Castration-Resistant Prostate Cancer

Abstract

Survival mechanisms elicited by CRPC C4-2B cells when treated with Enza may be blocked by inhibiting autophagy with clomipramine and metformin. Combination of Enza with saracatinib and autophagy modulators significantly reduced cell proliferation in the LNCaP GRP CRPC model. Further in vivo studies will provide evidences to make autophagy targeting as a supplement to cancer therapeutic regimes.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 2013
Accession Number
ADA597874

Entities

People

  • Christopher P Evans

Organizations

  • University of California

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Androgen Receptors
  • Androgens
  • Antidepressants
  • Apoptosis
  • Autophagy
  • Castration
  • Cell Line
  • Cell Physiological Processes
  • Department Of Defense
  • Inhibition
  • Inhibitors
  • Modulators
  • Neoplasms
  • Prostate
  • Prostate Cancer
  • Survival
  • Targeting

Fields of Study

  • Biology
  • Chemistry

Readers

  • Molecular and Cellular Biology
  • Prostate Cancer Biology.