Targeting Neuroendocrine Differentiation for Prostate Cancer Radiosensitization

Abstract

Radiotherapy (RT) is an important primary treatment for low-risk prostate cancer and the standard treatment for high-risk prostate cancer when combined with hormone therapy. Despite that many patients can be cured by RT, several studies suggest that approximately 10% of patients with low-risk cancer and up to 30-60% of patients with high-risk cancer experience biochemical recurrence within five years after RT, among them 20% of patients die in 10 years. Neuroendocrine differentiation (NED) is a process by which prostate cancer cells transdifferentiate into neuroendocrine-like (NE-like) cells. NED is associated with disease progression and treatment failure. Based on our finding that the transcription factor cAMP response element (CREB) is responsible for fractionated ionizing radiation (FIR)-induced NED, we hypothesized that targeting neuroendocrine differentiation can sensitize prostate cancer cells to radiation. We proposed two CREB targeting strategies as a model system to test our hypothesis. During the first year of grant support, we have established multiple stable and doxycycline/tetracycline-inducible cell lines that express short hairpin RNAs (shRNAs) to knock down CREB or express ACREB, a dominant negative mutant of CREB. We have examined the effect of ACREB expression on FIR-induced cell death in LNCaP cells, and found that induction of ACREB during the first two weeks (weeks 1-2), the second two weeks (weeks 3-4), or the entire four weeks (weeks 1-4) efficiently increased FIR-induced cell death and inhibited the extent of NED in survival cells. Further, clonogenic assays have also showed that ACREB expression sensitized LNCaP cells to radiation in a dose-dependent manner. In support of this notion, CREB knockdown also sensitized LNCaP cells to radiation in clonogenic assays.

Open PDF

Document Details

Document Type
Technical Report
Publication Date
Oct 01, 2014
Accession Number
ADA613326

Entities

People

  • Chang-deng Hu
  • Gyeon Oh
  • Xuehong Deng

Organizations

  • Purdue University

Tags

DTIC Thesaurus Topics

  • Biochemistry
  • Biological Sciences
  • Biomedical Research
  • Cell Biology
  • Cell Line
  • Cell Physiological Processes
  • Cells
  • Chemistry
  • Diseases And Disorders
  • Health Services
  • Ionizing Radiation
  • Molecular Biology
  • Neoplasms
  • Protein-Protein Interactions
  • Proteins
  • Therapy
  • Transcription Factors

Fields of Study

  • Biology
  • Medicine

Readers

  • Prostate Cancer Biology.